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العنوان
study of gastro-duodenal angiodysplasia in patients with chronoic renal failure/
الناشر
hassan galal abd el salam,
المؤلف
abd el salam,hassan galal
هيئة الاعداد
باحث / حسن جلال عبدالعظيم
مشرف / اكرام مصطفى الاسيوطى
مشرف / المتولى الشهاوى
مشرف / محمد سالم
مشرف / محمد شوقى
الموضوع
internal medicine
تاريخ النشر
1995 .
عدد الصفحات
194p.:
اللغة
الإنجليزية
الدرجة
الدكتوراه
التخصص
الطب الباطني
تاريخ الإجازة
1/1/1995
مكان الإجازة
جامعة بنها - كلية طب بشري - باطنه
الفهرس
Only 14 pages are availabe for public view

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from 210

Abstract

This study was done on twenty five patients with chronic renal failure on regular hemodialysis.
The mean value of their ages was 44.1 ± 8.5 years and the mean value of their duration of dialysis
was 2.8 ± 2.2 years. The following findings were reported in this study:
1-Gastro-duodenal angiodysplasia was found in 32% of the study group at the start of work and 62.5% of them were bleeding (melaena ±
hamatemesis) 2-Cases with angiodysplasia showed microcytic
hypochromic anemia more than cases without it
due to more risk recurrent bleeding.
3-Cases with angiodysplasia showed no
bleeding tendency than cases without it
more
4-Cases with angiodysplasia showed no
significant difference ln biochemical
parameters measured including: fasting blood
sugar, blood urea, serum creatinine, serum
calcium and serum phosphate.
5-Cases with angiodysplasia showed higher serum
aluminum levels (evidenced by BAL) and higher
- 147-
body stores of aluminum (evidenced by DAL)
than cases without it.
6-Prevalence of angiodysplasia is higher with
higher serum aluminum level and with longer
duration of dialysis.
7-Decreasing serum aluminum level by administration
of Dfo led to significant improvement
of angiodysplasia as it was detected in l2%
of the studied group at the end of the work.
So, it is concluded that:
A} Angiodysplasia of the upper gastrointestinal
tract is an important cause of upper gastrointestinal
hemorrhage in patients with chronic
renal failure on regular hemodialysis.
B) Aluminum accumulation in the body of these patients may be responsible for the development of angiodysplasia.