الفهرس 
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Abstract
There was significant positive correlation between AP-36 and BMI in PCOS group (r=0.529, P<0.001). There was non-significant fair positive correlation between AP-36 and age and LH/FSH ratio and non-significant weak correlation with FBG, fasting insulin, HOMA-IR, TG, TC, HDL, LDL, FSH, LH, fT, E2, prolactin, fT4 and TSH.There was significant positive correlation between AP-36 and BMI in subgroup Ia (r=0.536, P=0.008). There was non-significant fair positive correlation between AP-36 and age and HOMA-IR and non-significant weak correlation with FBG, fasting insulin, TG, TC, HDL, LDL, FSH, LH, LH/FSH ratio, fT, E2, prolactin, fT4 and TSH.There was non-significant fair positive correlation between AP-36 and BMI and LH/FSH ratio, non-significant fair negative correlation between AP-36 and FBG, fasting insulin and HOMA-IR and non-significant weak correlation with age, TG, TC, HDL, LDL, FSH, LH, fT, E2, prolactin, fT4 and TSH.All PCOS patients and obese subgroup exhibit increased AP-36 level with higher fasting insulin level, HOMA-IR, LH level, LH/FSH ratio and fT level, but lower FSH and E2 levels than controls, while non-obese PCOS patients exhibit increased TG level, LH/FSH ratio and fT but lower FSH and E2 levels than matched controls. Moreover obese PCOS exhibit increased AP-36 level, fasting insulin level and HOMA-IR than non-obese PCOS.
Serum AP-36 level is positively correlated with BMI in patients with PCOS.
According to our data, AP-36 is not directly implicated in the pathogenesis of PCOS, but it might be involved as an adipokine that is affected by BMI.